August 4, 2025

Supplementary Figure S6 from VAV2 Drives EGFR-Mediated Rac1 Responses in Prostate Cancer

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VAV2 knock-out (KO) prostate cancer cells exhibit reduced Rac1 activation.
EGF treatment leads to significant activation of both Rac1 and Cdc42 in prostate cancer cells.
The experiment demonstrates the involvement of VAV2 in EGFR-mediated signaling pathways.
Understanding VAV2's role may help develop targeted therapies for prostate cancer.

Implication

Figure shows EGF induces Rac1 and Cdc42 activation in VAV2 KO prostate cancer cells, suggesting VAV2's role in signaling pathways.

Figure S6. EGF-induced Rac1 and Cdc42 activation in VAV2 KO prostate cancer cells.

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Baker et al. (2025) studied this question.

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Figure shows EGF induces Rac1 and Cdc42 activation in VAV2 KO prostate cancer cells, suggesting VAV2's role in signaling pathways.

Figure S6. EGF-induced Rac1 and Cdc42 activation in VAV2 KO prostate cancer cells.