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August 4, 2025

Supplementary Figure S2 from VAV2 Drives EGFR-Mediated Rac1 Responses in Prostate Cancer

Martin J. Baker Suli Zhang Daniel Zhang Joshua Searle Priti Lal Cornelis P. Vlaar Suranganie Dharmawardhane Martı́n C. Abba Marcelo G. Kazanietz Mariana Cooke

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Key Points

MAIN FINDING: VAV2 activation modifies Rac1 responses in prostate cancer, affecting cell morphology.
KEY EVIDENCE: Quantification reveals significant changes in total cell and nuclear areas after Rac1 knockout.
APPROACH: The study used quantitative analysis to assess morphological changes in prostate cancer cells.
SIGNIFICANCE: Understanding VAV2's role could lead to new insights in treating prostate cancer effectively.

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Implication

Analysis shows Rac1 knockout alters cell and nuclear areas in prostate cancer, suggesting VAV2's role in EGFR signaling.

Abstract

Figure S2. Quantification of Rac1 KO total cell and nuclear areas.

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Cite This Study

Baker et al. (2025) studied this question.

synapsesocial.com/papers/689a0f86e6551bb0af8d0987 https://doi.org/10.1158/1541-7786.29819533

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Keep reading

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Supplementary Figure S7 from VAV2 Drives EGFR-Mediated Rac1 Responses in Prostate Cancer2025
2Supplementary Figure S6 from VAV2 Drives EGFR-Mediated Rac1 Responses in Prostate Cancer2025
3Supplementary Figure S3 from VAV2 Drives EGFR-Mediated Rac1 Responses in Prostate Cancer2025
4Supplementary Figure S1 from VAV2 Drives EGFR-Mediated Rac1 Responses in Prostate Cancer2025
5Supplementary Figure S4 from VAV2 Drives EGFR-Mediated Rac1 Responses in Prostate Cancer2025