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September 3, 2025Open Access

Supplementary Data 2 from HuR-Regulated Extracellular Vesicles Promote Endothelial Cell Remodeling in Pancreatic Cancer

Jennifer M. Finan Yifei Guo Alexandra Q. Bartlett Kevin Hawthorne Matthew Reyer Margaret Haerr Olayinka Lamikanra Hen Halamish Valerie Calvert Canping Chen Zheng Xia Emanuel F. Petricoin Rosalie C. Sears Katelyn T. Byrne Jonathan R. Brody

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Key Points

HuR-regulated extracellular vesicles enhance endothelial cell remodeling in pancreatic cancer.
Proteomic data shows significant differences in EV cargo between wild-type and HuR knockout models.
The study provides insight into how these vesicles could influence cancer progression and therapy.
Targeting the interactions of these vesicles may lead to innovative treatment strategies for pancreatic cancer.

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Implication

Proteomic analysis reveals HuR KO alters vesicle cargo in pancreatic cancer, suggesting new therapeutic targets.

Abstract

Supplementary Data 2. Proteomic data from WT vs. HuR KO EV cargo

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Cite This Study

Finan et al. (2025) studied this question.

synapsesocial.com/papers/68c183f09b7b07f3a060fa76 https://doi.org/10.1158/2767-9764.30038881

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Supplementary Data 1 from HuR-Regulated Extracellular Vesicles Promote Endothelial Cell Remodeling in Pancreatic Cancer2025
2Supplementary Data 3 from HuR-Regulated Extracellular Vesicles Promote Endothelial Cell Remodeling in Pancreatic Cancer2025
3Supplementary Figure S1 from HuR-Regulated Extracellular Vesicles Promote Endothelial Cell Remodeling in Pancreatic Cancer2025
4Supplementary Figure S6 from HuR-Regulated Extracellular Vesicles Promote Endothelial Cell Remodeling in Pancreatic Cancer2025
5Supplementary Figure S3 from HuR-Regulated Extracellular Vesicles Promote Endothelial Cell Remodeling in Pancreatic Cancer2025