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April 29, 2024AllergyOpen Access

Neutrophil‐driven and interleukin‐36γ‐associated ocular surface inflammation in chronic Stevens–Johnson syndrome

Madhuri Amulya Koduri Tejaswini Pingali Deeksha Prasad Vijay Singh Swati Singh Swapna S. Shanbhag Sayan Basu Vivek Singh

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Abstract

This study aims to elucidate the tear proteome and understand the underlying molecular mechanisms involved in the ocular complications following Stevens-Johnson syndrome/toxic epidermal necrolysis (SJS/TEN).

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Cite This Study

Koduri et al. (2024) studied this question.

synapsesocial.com/papers/68e6d055b6db64358764de25 https://doi.org/10.1111/all.16126

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1An update on our understanding of host defense mechanisms at the ocular surface; innate immune dysregulation at the ocular surface: lessons from Stevens–Johnson syndrome and related inflammatory disordersReview · 2026
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3Ocular Complications in stevens-Johnson Syndrome and Toxic Epidermal Necrolysis: A Comprehensive Review2025
4Chronic Ocular Complications in Stevens–Johnson Syndrome/Toxic Epidermal Necrolysis: Clinical Features and Surgical Management in a Brazilian Tertiary Center2025
5Immunological Interplay at the Ocular Surface of Stevens-Johnson Syndrome, Ocular Cicatricial Pemphigoid, and Ocular Graft Versus Host DiseaseOriginal Research · 2026