Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
March 11, 2024PLoS PathogensOpen Access

Hepatitis B virus e antigen induces atypical metabolism and differentially regulates programmed cell deaths of macrophages

View Full Paper
Ask AI
Bookmark
Share

Authors

YLYumei LiCWChristine WuJLJi‐Young Lee

Discussion

Loading...

Member takes

Overview

Key Points

Key points are not available for this paper at this time.

Cite This Study

Li et al. (2024) studied this question.

synapsesocial.com/papers/68e74952b6db6435876c2153https://doi.org/10.1371/journal.ppat.1012079
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1HBV induces immunosuppressive macrophages via metabolic and epigenetic reprogramming to facilitate the establishment of chronic persistent infection2026
  2. 2Activation of the cGAS-STING pathway by viral dsDNA leading to M1 polarization of macrophages mediates antiviral activity against hepatitis B virus2024 · 19 citations
  3. 3The hijacking of HBV by small extracellular vesicles inhibits M1 macrophages to facilitate immune evasion2024 · 6 citations
  4. 4Metabolic associated fatty liver disease induced hepatocyte apoptosis to eliminate hepatitis B virus2026
  5. 5Interferon‐γ<sup>+</sup> Th1 activates intrahepatic resident memory T cells to promote HBsAg loss by inducing M1 macrophage polarization2024 · 14 citations