The hijacking of HBV by small extracellular vesicles inhibits M1 macrophages to facilitate immune evasion

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1. 1Proteome profiling of extracellular vesicles-derived from hepatitis B virus-infected hepatocellular carcinoma cell lines identifies PDCD11 as a carrier of viral RNAs2025
2. 2Hepatitis B virus e antigen induces atypical metabolism and differentially regulates programmed cell deaths of macrophages2024 · 14 citations
3. 3Activation of the cGAS-STING pathway by viral dsDNA leading to M1 polarization of macrophages mediates antiviral activity against hepatitis B virus2024 · 19 citations
4. 4Macrophages treated with interferons induce different responses in lymphocytes via extracellular vesicles2024 · 5 citations
5. 5HBV induces immunosuppressive macrophages via metabolic and epigenetic reprogramming to facilitate the establishment of chronic persistent infection2026