Observational analysis shows nicorandil reduces inflammatory response and oxidative stress in myocardial infarction, suggesting it may enhance cardiac function.
Nicorandil attenuates myocardial inflammation, oxidative stress and myocardial fibrosis levels by activating ATP-sensitive potassium channels (K-ATP), thereby improving post-MI cardiac remodeling and preserving cardiac function.
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Huang et al. (2025) studied this question.
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