Cardiac inflammation plays a pathophysiological role in HFNEF by promoting extracellular matrix accumulation and subsequent diastolic dysfunction.
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Supports inflammation-ECM pathway in HFNEF diastolic dysfunction; hypothesis-generating and should not yet change practice.
Cardiac inflammation contributes to diastolic dysfunction in HFNEF by triggering the accumulation of ECM.
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Westermann et al. (2010) studied this question.
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