Cardiac Magnetic Resonance in Heart Failure: Diagnostic and Prognostic Assessments

Figures

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  Figure 1. Dilated cardiomyopathy (LMNA mutation) with diffuse LGE showing «ring-like» pattern.

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  Figure 2. Short axis views of left ventricular acute myocarditis. Myocardial aedema in T2 weighted sequence (A); subepicardial LGE (B).

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  Figure 3. Short axis (A) and long axis (B) views of left ventricular LGE patchy distribution in sarcomeric hypertrophic cardiomyopathy.

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  Figure 4. Short axis views in Fabry disease. Cine sequence showing left ventricular hypertrophy (A); typical LGE distribution in mid-wall area of infero-lateral segment (B; arrow); global reduction of native T1-mapping (purple areas; global native T1: 943 ms) except for infero-lateral wall (C).

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  Figure 5. Transthyretin amyloidosis. Cine-imaging showing left ventricular hypertrophy (A). Diffuse patchy LGE (B). Very high T1 mapping and ECV values (1190 ms and 45%, respectively) (C).

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  Figure 6. Cardiac sarcoidosis. CMR T2 imaging showing septal oedema (A). Septal LGE (B). Corresponding FDG-PET imaging showing high septal inflammation (C).

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  Figure 7. Graphs of the relaxation of T2* values over time to compute T2* of the liver (A) and myocardium (B). T2* values are consistent with severe iron overload of the liver (0.7 ms) and myocardium (8 ms). T1 mapping values are also very low (about 770 ms) (C).

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  Figure 8. Arrhythmogenic cardiomyopathy. Short-axis view, black-blood flow, showing right ventricle and interventricular septum fatty infiltration (A). Four chamber view showing LGE in both ventricular chambers (B).