Key result
Thrombotic LCX occlusion in dogs induced NSVT in 90%, VT in 60%, and VF in 50%, with longer activation times identifying VTs that degenerated into VF (169 vs 81 msec).
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Hypothesis-generating in canine ischemia; leaves open activation time for VF risk stratification in human ACS.
Zhang et al. (2000) studied Ventricular arrhythmias induced by thrombotic coronary occlusion (n=10). Thrombotic coronary occlusion was evaluated on Incidence of NSVT, VT, and VF, and activation time. Thrombotic LCX occlusion in dogs induced NSVT in 90%, VT in 60%, and VF in 50%, with longer activation times identifying VTs that degenerated into VF (169 vs 81 msec).
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