Why the study?
Does atorvastatin therapy during the peri-infarct period improve left ventricular function and attenuate remodeling after myocardial infarction in a rat model?
Population
40 Female Fischer 344 rats subjected to permanent coronary occlusion.
Comparison
Oral atorvastatin 10 mg/kg/d by gavage daily for… vs Equivalent volume of vehicle daily for 7 weeks.
Design
Preclinical, Echocardiographic images analyzed off-line by a single blinded…
Follow-up
4 weeks after myocardial infarction
Key result
Atorvastatin therapy during the peri-infarct period significantly improved left ventricular ejection fraction (34.16% vs 24.30%, p<0.05) and limited adverse remodeling following myocardial infarction in rats.
Authors
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Hypothesis-generating for peri-infarct atorvastatin post-MI; leaves open translation to human practice.
Does atorvastatin therapy during the peri-infarct period improve left ventricular function and attenuate remodeling after myocardial infarction in a rat model?
Absolute Event Rate: 34.16% vs 24.3%
p-value: p=<0.05
In a rat model of myocardial infarction, peri-infarct atorvastatin therapy significantly improved left ventricular function and reduced adverse remodeling, fibrosis, and apoptosis, independent of infarct size.
Tang et al. (2011) studied Myocardial infarction (n=40). Atorvastatin vs. Vehicle (water) was evaluated on Left ventricular ejection fraction (LVEF) at 4 weeks (p=<0.05). Atorvastatin therapy during the peri-infarct period significantly improved left ventricular ejection fraction (34.16% vs 24.30%, p<0.05) and limited adverse remodeling following myocardial infarction in rats.
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