Key result
Defective excitation-contraction coupling in heart failure may result from altered density or function of proteins relevant for calcium homeostasis.
Design
Review
Authors
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Supports research on calcium cycling proteins in heart failure; leaves open clinical translation pending prospective trials.
Altered density or function of calcium cycling proteins may contribute to defective excitation-contraction coupling in heart failure.
Hadri et al. (2011) conducted a review in Heart failure. Defective excitation-contraction coupling in heart failure may result from altered density or function of proteins relevant for calcium homeostasis.
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