This review emphasizes the unique embryologic, morphological, and physiologic properties of the right ventricle and the need to understand RV-specific mechanisms of fibrosis rather than extrapolating from left ventricular models.
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RV fibrosis in pulmonary hypertension warrants RV-specific investigation beyond LV models; leaves open targeted antifibrotic strategies for right ventricular failure.
Nikolaos G. Frangogiannis (2017) studied this question.
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