In a rat model of myocardial infarction, the progression to overt heart failure is associated with a metabolic shift characterized by upregulation of GLUT-1 and downregulation of MCAD.
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Metabolic shift in post-MI rat HF supports further substrate-target investigation; leaves open human relevance and therapeutic impact.
Nathalie Rosenblatt‐Velin (2001) studied this question.
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