Population
11 index patients with primary hypertriglyceridemia and their 70 first-degree relatives (total n=81)
Design
Cross-sectional
Authors
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Supports genetic control of VLDL-TG catabolism; hypothesis-generating for familial hypertriglyceridemia pending prospective studies.
This study demonstrates that the catabolism of VLDL-TG is under genetic control, whereas its production rate is primarily related to obesity, providing mechanistic insights into familial hypertriglyceridemia.
Sane et al. (1988) studied this question.
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