Why the study?
Does exogenous thrombin alter vascular reactivity differently in ischemic compared to nonischemic canine coronary arteries?
Does exogenous thrombin alter vascular reactivity differently in ischemic compared to nonischemic canine coronary arteries?
Endothelial damage from ischemia shifts the coronary vascular response to thrombin from vasodilatation to vasoconstriction, suggesting a mechanism for coronary vasospasm.
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May promote thrombin-mediated coronary spasm after endothelial ischemia; leaves open relevance to human vasospasm.
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David D. Ku (1982) studied this question.
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