Key result
Spontaneously hypertensive rats had significantly more ultradian power than normotensive and renovascular hypertensive rats, and lacked the circadian modulation of ultradian power seen in normotensives.
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SHR-specific ultradian BP features extend variability research beyond circadian rhythms; leaves open human applicability.
Holstein‐Rathlou et al. (1995) studied Hypertension. Hypertension models (SHR and 2K,1C) vs. Normotensive Sprague-Dawley rats was evaluated on Patterns in mean arterial pressure (power spectra in ultradian, circadian, and infradian domains). Spontaneously hypertensive rats had significantly more ultradian power than normotensive and renovascular hypertensive rats, and lacked the circadian modulation of ultradian power seen in normotensives.
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