Key result
Fibrinogen plays a central role in thrombosis, inflammation, and the renin-angiotensin system, contributing more significantly to premature coronary artery disease in women than in men.
Why the study?
Sufficiently powered clinical and experimental studies assessing multipathogenic, sex-related differences in coronary artery disease formation and progression across different age periods are lacking.
Design
Review
Authors
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Fibrinogen's greater role in women's premature CAD warrants caution in risk models; leaves open sex-specific prospective trials.
Fibrinogen and its interaction with inflammation and the renin-angiotensin system may drive sex-specific differences in the pathogenesis of premature coronary artery disease in women.
Kryczka et al. (2021) conducted a review in Premature Coronary Artery Disease. Fibrinogen vs. Men was evaluated. Fibrinogen plays a central role in thrombosis, inflammation, and the renin-angiotensin system, contributing more significantly to premature coronary artery disease in women than in men.
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