Key result
Circulating catecholamines modulate platelet reactivity via alpha(2A)-adrenoceptors, potentially mediating the interplay between stress, sympathoadrenal activation, and cardiovascular diseases.
Catecholamine-induced platelet activation provides a mechanistic link between stress, sympathoadrenal hyperactivation, and the pathogenesis of cardiovascular diseases.
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Suggests catecholamines modulate platelet reactivity in hypertension and atherosclerosis; leaves open clinical relevance pending interventional data.
Anfossi et al. (1996) conducted a review in Cardiovascular diseases (atherosclerosis and arterial hypertension). Catecholamines was evaluated. Circulating catecholamines modulate platelet reactivity via alpha(2A)-adrenoceptors, potentially mediating the interplay between stress, sympathoadrenal activation, and cardiovascular diseases.