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January 1, 2002Thrombosis and Haemostasis

Exposing platelets to a high shear stress of 100 dyn/cm2 for 2.5 seconds followed by low shear induced aggregation with significantly less CD62P expression than ADP treatment.

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Population

Platelets (in vitro model of arterial stenosis)

Comparison

Exposure to a shear profile containing both high… vs Platelets treated with ADP

Design

Preclinical

Key result

Exposing platelets to a high shear stress of 100 dyn/cm2 for 2.5 seconds followed by low shear induced aggregation with significantly less CD62P expression than ADP treatment.

Authors

JZJian-Ning ZhangABAngela L. BergeronQYQinghua Yu

Discussion

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Overview

Questions prolonged high-shear in vitro platelet assays for arterial stenosis; leaves open the thrombotic contribution of post-stenotic low flow.

Structured PICO

P
Population
Platelets (in vitro model of arterial stenosis)
I
Intervention
Exposure to a shear profile containing both high (100 dyn/cm2 for 2.5 s) and low shear segments
C
Comparator
Platelets treated with ADP (for activation comparison)
O
Outcome
Platelet aggregation and activation (CD62P expression)surrogate

Post-stenosis low flow enhances shear-induced platelet aggregation, which appears to be a mechanical crosslinking process requiring minimal platelet activation.

Cite This Study

Zhang et al. (2002) studied this question. Complex pattern of shear stress (high shear followed by low shear) vs. ADP treatment was evaluated on Platelet aggregation and CD62P expression. Exposing platelets to a high shear stress of 100 dyn/cm2 for 2.5 seconds followed by low shear induced aggregation with significantly less CD62P expression than ADP treatment.

synapsesocial.com/papers/6a6d6a35f44fa9f079dbaaaehttps://doi.org/10.1055/s-0037-1613308
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