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This commentary discusses a preclinical study demonstrating that renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in a rat model of heart failure.
We comment on the publication of a paper in which Brazilian investigators evaluate the anticontractile response of perivascular adipose tissue (PVAT) in experimental heart failure (HF) induced in rats by occlusion of a coronary artery.
Carlos M. Ferrario (2021) studied this question.
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