Population
Mammalian cells
Comparison
Treatments causing ER accumulation of caveolin-1 vs Normal caveolin-1 expression and localization
Design
Preclinical
Authors
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Should not yet alter clinical lipid management; hypothesis-generating for caveolin-1 ER exit signals in lipid droplet targeting.
Accumulation of caveolin-1 in the endoplasmic reticulum redirects the protein to lipid storage droplets, indicating a need for rapid exit from the ER to maintain normal localization.
Ostermeyer et al. (2001) studied this question.
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