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March 6, 2003Circulation Research

Is Depressed Myocyte Contractility Centrally Involved in Heart Failure?

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Why the study?

Does depressed myocyte contractility initiate the heart failure syndrome and drive its progression?

Design

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Authors

SHSteven R. HouserKMKenneth B. Margulies

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Overview

Human data leave contractility's causal role in HF initiation unproven; extends animal signals but requires mechanistic trials before guiding inotrope strategies.

Key Points

Key points are not available for this paper at this time.

Cite This Study

Houser et al. (2003) studied this question.

synapsesocial.com/papers/6a704268ac440176ef28e7ebhttps://doi.org/10.1161/01.res.0000060027.40275.a6
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Congestive Heart Failure: Fifty Years of Progress2000 · 440 citations
  2. 2Adenoviral gene transfer of SERCA2a improves left-ventricular function in aortic-banded rats in transition to heart failure2000 · 538 citations
  3. 3Dilated cardiomyopathy in transgenic mice expressing a dominant-negative CREB transcription factor in the heart.1998 · 249 citations
  4. 4AKAP-Mediated Targeting of Protein Kinase A Regulates Contractility in Cardiac Myocytes2001 · 153 citations
  5. 5Local Control Models of Cardiac Excitation–Contraction Coupling1999 · 278 citations