Population
Human umbilical vein endothelial cells, including VE-cadherin knock-out cells and cells re-expressing…
Design
Preclinical
Authors
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Epac1 may modulate endothelial barrier function in vitro; leaves open therapeutic targeting in vascular leak syndromes.
Epac1 controls VE-cadherin-mediated cell junction formation and induces reorganization of the actin cytoskeleton in human endothelial cells.
Kooistra et al. (2005) studied this question.
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