Why the study?
Does intravenous injection of hypertonic NaCl solution stimulate pulmonary C-fibers in anesthetized dogs?
Does intravenous injection of hypertonic NaCl solution stimulate pulmonary C-fibers in anesthetized dogs?
Intravenous injection of hypertonic solutions evokes reflex bradycardia and hypotension principally through the stimulation of pulmonary C-fibers.
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Hypertonic NaCl stimulates pulmonary afferents mediating reflexes in dogs; leaves open translation to human intravenous use.
Intravenous injection of hypertonic NaCl solution evokes reflex bradycardia and hypotension, effects thought to result from stimulation of afferent vagal endings in the lungs. To identify the afferents responsible for these effects, we recorded vagal impulses arising from endings in the lungs and lower airways of anesthetized dogs and examined the response to injection of hypertonic solutions into the pulmonary circulation. Injection of 4,800 mmol/l NaCl solution (1 ml/kg) stimulated 39 of 49 pulmonary C-fibers, their impulse frequency increasing 35-fold. Stimulation was concentration dependent, the minimum effective concentration being between 1,200 and 4,800 mmol/l. Rapidly adapting receptors were also stimulated in a concentration-dependent manner, 35 of 41 receptors being stimulated by 4,800 mmol/l NaCl solution, firing increasing fivefold. Bronchial C-fibers were not stimulated by injection into the pulmonary circulation but were by injection into the bronchial artery. Hypertonic urea solutions had qualitatively similar but smaller effects on pulmonary C-fibers and rapidly adapting receptors. The results suggest that the reflex effects of intravenous injection of hypertonic solutions result principally from stimulation of pulmonary C-fibers.
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Pisarri et al. (1991) studied this question.
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