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May alter catecholamine response interpretation post-denervation; leaves open relevance to human cardiovascular autonomic disorders.
From observations published during the last year or so, it would appear that post- ganglionic sympathetic denervation usually potentiates the response of effector organs to noradrenaline more than their response to adrenaline. This has been shown for the nictitating membrane of the cat by Bulbring and Burn (1949) and Lockett (1950a), and for the pupil dilator muscle of the cat by Burn and Hutcheon (1949). Lockett (1950b) found that in dogs thoracic and lumbar sympathectomy on the one hand potentiated the pressor response to noradrenaline more than that to adrenaline, but, on the other, increased the chronotropic action of adrenaline more than of noradrenaline.
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Innes et al. (1951) studied this question.
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