Population
Knock-in mice carrying the BDNFVal66Met human polymorphism, wild-type mice, WT cells, and human subjects.
Comparison
SIRT1 activation, SIRT1 inhibition, or… vs Wild-type mice, WT cells, or untreated controls.
Design
Preclinical
Authors
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Animal data on BDNFVal66Met link psychiatric traits to CVD; leaves open human translation and clinical relevance.
The BDNFVal66Met polymorphism, associated with depression, induces a prothrombotic state and increases AMI risk, providing a mechanistic link between psychiatric conditions and cardiovascular disease.
Amadio et al. (2015) studied this question.
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