In a 2D model of anisotropic cardiac muscle, directional changes in the maximum rate of rise correspond to small modifications of extracellular potentials, while changes in the time constant of the foot are associated with propagation velocity.
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Small extracellular signal changes accompany rate-of-rise shifts in 2D models; leaves open translation to human conduction.
Roberge et al. (1986) studied this question.
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