August 15, 2001Journal of Virology

A Human Rotavirus with Rearranged Genes 7 and 11 Encodes a Modified NSP3 Protein and Suggests an Additional Mechanism for Gene Rearrangement

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Authors

EGElyanne GaultAssistance Publique – Hôpitaux de Paris NSNathalie SchnepfHôpital Saint-Louis DPDidier PoncetTravaux et Recherches Archéologiques sur les Cultures, les Espaces et les Sociétés

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Implication

Gene rearrangement yielding dual ORFs may signal chronic rotavirus persistence in immunodeficient children; leaves open mechanisms of viral evolution and clinical relevance.

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Key points are not available for this paper at this time.

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Gault et al. (2001) studied this question.

synapsesocial.com/papers/6a75333a756843cacbb1ede0 https://doi.org/10.1128/jvi.75.16.7305-7314.2001

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Implication

Gene rearrangement yielding dual ORFs may signal chronic rotavirus persistence in immunodeficient children; leaves open mechanisms of viral evolution and clinical relevance.

A Human Rotavirus with Rearranged Genes 7 and 11 Encodes a Modified NSP3 Protein and Suggests an Additional Mechanism for Gene Rearrangement

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Implication

Key Points

Cite This Study

A Human Rotavirus with Rearranged Genes 7 and 11 Encodes a Modified NSP3 Protein and Suggests an Additional Mechanism for Gene Rearrangement

Discussion

Member takes

Implication

Key Points

Cite This Study

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