Why the study?
Is defective suppressor immune regulatory function present in patients with idiopathic congestive cardiomyopathy and rheumatic heart disease compared to controls?
Population
Patients with idiopathic congestive cardiomyopathy and rheumatic heart disease, and control subjects
Comparison
In vitro evaluation of suppressor immune… vs Control subjects
Design
Case-control
Authors
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Does not support defective suppressor function as a primary mechanism in these cardiomyopathies; leaves open alternative immunoregulatory pathways for targeted study.
Is defective suppressor immune regulatory function present in patients with idiopathic congestive cardiomyopathy and rheumatic heart disease compared to controls?
The study demonstrates that a chronic immunoregulatory defect in suppressor cell function is not a primary mechanism in idiopathic congestive cardiomyopathy or rheumatic heart disease.
Anderson et al. (1981) studied this question.
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