Key result
The mechanisms of arrhythmias in diabetes are complex and involve changes in blood sugar levels, cardiac autonomic neuropathy, remodeling, fibrosis, oxidative stress, and metabolic changes.
Why the study?
The mechanism of arrhythmias in patients with diabetes mellitus is complex, unique, and involves multiple distinct pathophysiological pathways.
This narrative review highlights the complex, multifactorial mechanisms—including autonomic neuropathy, structural remodeling, and glycemic fluctuations—that predispose patients with diabetes mellitus to cardiac arrhythmias.
Complex arrhythmia mechanisms in diabetes warrant holistic risk assessment; leaves open dominant pathway identification for targeted therapies.
Diabetes mellitus is a metabolic disorder characterized by an increase in blood sugar levels. The mechanism of arrhythmias in patients with diabetes mellitus is complex and unique. This is an independent factor in the occurrence of arrhythmias through several slightly different mechanisms in non-diabetics. Hyperglycemia, hypoglycemia, remodeling, autonomic neuropathic dysfunction, fibrosis, and oxidative changes, and metabolic stress play an important role in the occurrence of arrhythmias in diabetes. This narrative review was at least to describe the mechanism of arrhythmia in diabetes mellitus.
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Mezal et al. (2022) conducted a review in Diabetes mellitus and arrhythmias. Diabetes mellitus was evaluated. The mechanisms of arrhythmias in diabetes are complex and involve changes in blood sugar levels, cardiac autonomic neuropathy, remodeling, fibrosis, oxidative stress, and metabolic changes.
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