Population
Wistar rats with diabetic cardiomyopathy induced by low-dose streptozotocin-high-fat diet, patients with…
Comparison
miR-30c overexpression and inhibition (in vitro) vs Control rats and normal glucose-treated…
Design
Preclinical
Follow-up
12 weeks (for rat model)
Authors
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Identifies miR-30c-Cdc42/Pak1 as potential target in diabetic cardiomyopathy models; hypothesis-generating and requires in vivo validation.
Downregulation of miR-30c mediates prohypertrophic effects of hyperglycemia in diabetic cardiomyopathy by upregulating Cdc42 and Pak1 genes, identifying a potential therapeutic target.
Raut et al. (2015) studied this question.
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