Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
April 1, 1974Circulation ResearchOpen Access

Depressed Fatty Acid and Acetate Oxidation and Other Metabolic Defects in Homogenates from Hearts of Hamsters with Hereditary Cardiomyopathy

View Full Paper
Ask AI
Bookmark
Share

Authors

KKK. J. KakoMTM. ThorntonHHH. Alexander Heggtveit

Discussion

Loading...

Member takes

Overview

No immediate clinical implications for cardiomyopathy; leaves open metabolic contributions to human disease progression.

Key Points

Key points are not available for this paper at this time.

Cite This Study

Kako et al. (1974) studied this question.

synapsesocial.com/papers/6a83871fdaf812a7fbcd7cd4https://doi.org/10.1161/01.res.34.4.570
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Hamster cardiomyopathy. A defect in oxidative phosphorylation in the cardiac interfibrillar mitochondria.1982 · 117 citations
  2. 2Defective Oxidative Phosphorylation in Hereditary Myocardiopathy in the Syrian Hamster1968 · 38 citations
  3. 3Lipid content and fatty acid composition of heart and muscle of the BIO 82.62 cardiomyopathic hamster1976 · 25 citations
  4. 4Carnitine derivatives in hereditary cardiomyopathic animals.1985 · 6 citations
  5. 5Myocardial subcellular fractionation studies on cardiomyopathic Syrian hamsters1979 · 13 citations