Key result
Cardiomyopathic hamsters exhibit a mitochondrial oxidative defect characterized by significantly decreased state 3 ADP-stimulated respiration confined wholly to the interfibrillar mitochondria.
Authors
Loading...
Subcellular enzyme shifts in BIO 14.6 hamsters are hypothesis-generating; clinical translation to cardiomyopathy remains unproven.
Hoppel et al. (1982) studied Cardiomyopathy. Cardiomyopathy (BIO 14.6 strain) vs. Normal hamster hearts (controls) was evaluated on State 3 ADP-stimulated respiration in interfibrillar mitochondria. Cardiomyopathic hamsters exhibit a mitochondrial oxidative defect characterized by significantly decreased state 3 ADP-stimulated respiration confined wholly to the interfibrillar mitochondria.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: