Key result
BIO 82.62 cardiomyopathic hamsters showed significantly increased muscle cholesterol content at 67 and 108 days (P<0.01) and stored more saturated fatty acids in heart and muscle than normal controls.
Authors
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Indicates lipid dysregulation in genetic cardiomyopathy models; leaves open human relevance and therapeutic targeting.
p-value: p=<0.01
Barakat et al. (1976) studied Cardiomyopathy. BIO 82.62 cardiomyopathic strain vs. Age-matched normal animals was evaluated on Lipid content and fatty acid composition of heart and muscle (p=<0.01). BIO 82.62 cardiomyopathic hamsters showed significantly increased muscle cholesterol content at 67 and 108 days (P<0.01) and stored more saturated fatty acids in heart and muscle than normal controls.
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