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January 25, 2007Clinical and Experimental Pharmacology and Physiology

Angiotensin Ii‐induced Growth of Vascular Smooth Muscle Cells Is Associated With Modulation of Cell Surface Area and Platelet‐derived Growth Factor Receptor Expression

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Why the study?

Does the Angiotensin II inhibitor E4177 prevent abnormal growth of vascular smooth muscle cells in vitro and in vivo?

Population

Animal model of vein graft transplantation and primary culture of human aortic smooth muscle cells (HASMC)

Comparison

Angiotensin II inhibitor E4177 vs Control (untreated or Angiotensin II alone)

Design

Preclinical

Authors

SKSosei KumaEOEiji OkiTOToshihiro Onohara

Discussion

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Overview

E4177 counters Ang II-driven smooth muscle cell growth in vitro; hypothesis-generating for vascular remodeling, no clinical implications yet.

Structured PICO

Does the Angiotensin II inhibitor E4177 prevent abnormal growth of vascular smooth muscle cells in vitro and in vivo?

P
Population
Animal model of vein graft transplantation and primary culture of human aortic smooth muscle cells (HASMC)
I
Intervention
Angiotensin II inhibitor E4177
C
Comparator
Control (untreated or Angiotensin II alone)
O
Outcome
Vascular smooth muscle cell (VSMC) growth and cell surface area enlargementsurrogate

Angiotensin II promotes vascular smooth muscle cell growth and cell surface area enlargement, effects that can be blocked by the AngII inhibitor E4177.

Cite This Study

Kuma et al. (2007) studied this question.

synapsesocial.com/papers/6a8928dcf1df03d55b28bc6fhttps://doi.org/10.1111/j.1440-1681.2007.04535.x
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