Key result
Hyperkalemia can induce reversible Brugada pattern ECG changes, a rare manifestation that mimics Brugada syndrome.
Case Report (n=1)
Highlights the importance of recognizing hyperkalemia as a reversible cause of Brugada pattern ECG to guide appropriate clinical management and avoid misdiagnosis.
May warrant excluding hyperkalemia in Brugada-pattern ECGs; single case leaves open prevalence and validation.
Brugada syndrome (BrS) is an inherited disorder of cardiac ion channels characterized by peculiar ECG findings predisposing individuals to ventricular arrhythmias, syncope, and sudden cardiac death (SCD). Various electrolyte disturbances and ion channels blocking drugs could also provoke BrS ECG findings without genetic BrS. Clinical differentiation and recognition are essential for guiding the legitimate action. Hyperkalemia is well known to cause a wide variety of ECG manifestations. Severe hyperkalemia can even cause life threatening ventricular arrhythmias and cardiac conduction abnormalities. Most common ECG findings include peaked tall T waves with short PR interval and wide QRS complex. Since it is very commonly encountered disorder, physicians need to be aware of even its rare ECG manifestations, which include ST segment elevation and Brugada pattern ECG (BrP). We are adding a case to the limited literature about hyperkalemia induced reversible Brugada pattern ECG changes.
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Ameen et al. (2017) conducted a case report in Hyperkalemia induced Brugada phenocopy (n=1). Hyperkalemia was evaluated on Brugada pattern ECG changes. Hyperkalemia can induce reversible Brugada pattern ECG changes, a rare manifestation that mimics Brugada syndrome.
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