Why the study?
Does cardiac-specific overexpression of DGKzeta attenuate left ventricular remodeling and improve survival after myocardial infarction in a mouse model?
Does cardiac-specific overexpression of DGKzeta attenuate left ventricular remodeling and improve survival after myocardial infarction in a mouse model?
Cardiac-specific overexpression of DGKzeta suppresses left ventricular structural remodeling, reduces fibrosis, and improves survival after myocardial infarction in mice, suggesting it as a potential therapeutic target.
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May support DGKzeta as a post-MI target in mice; leaves open translation to human therapy.
Niizeki et al. (2006) studied this question.
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