Why the study?
Are peripheral venous platelet aggregates increased in patients with unstable angina or acute myocardial infarction compared to acutely ill noncardiac controls?
Are peripheral venous platelet aggregates increased in patients with unstable angina or acute myocardial infarction compared to acutely ill noncardiac controls?
The study found no significant evidence that circulating platelet aggregates initiate unstable angina, though they are increased in acute transmural myocardial infarction.
No takes yet. Share an insight, caveat, or question.
No support for platelet aggregates initiating unstable angina; leaves open their role in transmural MI pending prospective studies.
Willerson et al. (1977) studied this question.
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