Key result
The major structural protein of African swine fever virus, p73, assembles into large 50,000-kDa complexes indicative of viral capsid or matrix precursors on the endoplasmic reticulum membrane.
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Hypothesis-generating for ASFV antivirals; leaves open in vivo validation before any translational consideration.
Cobbold et al. (1998) studied African Swine Fever Virus (ASFV) infection. In vitro viral infection and metabolic labeling was evaluated on Subcellular distribution and complex formation of p73. The major structural protein of African swine fever virus, p73, assembles into large 50,000-kDa complexes indicative of viral capsid or matrix precursors on the endoplasmic reticulum membrane.
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