Why the study?
Myocardial infarction progresses to heart failure and myocardial fibrosis, and the gut microbiota plays an important role in ASCVD; this study investigated the protective effect of Lacticaseibacillus rhamnosus GG on isoprenaline-induced MI in rats.
Does Lacticaseibacillus rhamnosus GG prevent myocardial damage in an isoprenaline-induced myocardial infarction rat model?
Population
Four groups of seven rats
Comparison
Control vs ISO vs L. rhamnosus vs ISO + L. rhamnosus
Design
Preclinical animal study
Key result
Lacticaseibacillus rhamnosus GG decreased cardiac troponin-I, creatine kinase-MB, CRP, and TNF-alpha levels, increased IL-10 levels, and improved ECG parameters in rats with isoprenaline-induced MI.
Authors
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Hypothesis-generating for probiotic effects in experimental MI; human trials required before any clinical consideration.
Does Lacticaseibacillus rhamnosus GG prevent myocardial damage in an isoprenaline-induced myocardial infarction rat model?
Lacticaseibacillus rhamnosus GG demonstrates protective effects against isoprenaline-induced myocardial infarction in rats by reducing cardiac biomarkers and inflammation while improving ECG parameters.
Beyazçiçek et al. (2022) studied Isoprenaline-induced myocardial infarction (n=28). Lacticaseibacillus rhamnosus GG vs. Isoprenaline alone and control was evaluated on Myocardial damage markers (cardiac troponin-I, creatine kinase-MB, CRP, TNF-alpha, IL-10) and ECG changes. Lacticaseibacillus rhamnosus GG decreased cardiac troponin-I, creatine kinase-MB, CRP, and TNF-alpha levels, increased IL-10 levels, and improved ECG parameters in rats with isoprenaline-induced MI.
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