Key result
Hemorrhage-induced ACTH release involves oligosynaptic projections from atrial and carotid receptors to the hypothalamus.
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No immediate clinical implications for hemorrhage; leaves open targeted modulation of mapped brainstem-hypothalamic ACTH pathways.
Gann et al. (1977) studied Hemorrhage. Hemorrhage was evaluated on ACTH release. Central neural pathways mediating ACTH release in response to hemorrhage involve oligosynaptic projections from atrial and carotid receptors to the hypothalamus.
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