Key result
In myocardial infarcts, there is considerable regional variation in flmTc-PYP uptake and calcium accumulation, with maximal uptake in areas with residual perfusion and extensive necrosis.
The authors argue that the soluble state of Tc-PYP in infarcted tissue does not exclude a role for calcium phosphate binding, based on observed regional variations in uptake and calcium accumulation.
with myocardial infarcts produced by permanent coronary occlusion, we have shown considerable regional variation in flmTc-PYP uptake and calcium accumulation, with areas of maximal nmTc- PYP uptake in tissue samples with residual perfusion, extensive necrosis and significantly elevated calcium content.4 6We do not agree with Dr. Dewanjee's argument that the readily soluble state of 'mTc-PYP in infarcted tissue excludes a role for calcium phosphate binding.Significant amounts of calcium as well
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Patel et al. (1978) conducted a letter in Myocardial infarcts. In myocardial infarcts, there is considerable regional variation in flmTc-PYP uptake and calcium accumulation, with maximal uptake in areas with residual perfusion and extensive necrosis.
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