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September 5, 2025Open Access

Supplementary Figure S10 from RORγt Inhibition Reduces Protumor Inflammation and Decreases Tumor Growth in Experimental Models of Lung Cancer

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Analysis shows digoxin treatment does not alter NK17 and γ/δT17 cell infiltration in lung cancer models, implying potential therapeutic strategies.

Digoxin treatment does not change the infiltration of NK17, NKT17, and γ/δT17 cells into the lungs.
Findings indicate that RORγt inhibition effectively reduces tumor growth in experimental lung cancer.
Observational analysis in KrasG12D mice demonstrates a response to inflammation modulated by digoxin.
This suggests further investigation into RORγt pathways may uncover novel therapeutic targets.

Yamada‐Hara et al. (2025) studied this question.

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Analysis shows digoxin treatment does not alter NK17 and γ/δT17 cell infiltration in lung cancer models, implying potential therapeutic strategies.