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September 15, 2025

Figure S1 from Mitochondrial Calcium Uniporter Links Acetyl-CoA Metabolism and H3K27 Acetylation to Maintain Glioblastoma Stem Cells

Guangqin Liu Haoqian Zhang Siqi Chen Jun Gao Haixin Zhao Yan Dong Changwei Liu Xuechen Wei Ting Li Chang Lu Hai-Zhen Zhu Dingyi Lu Shiyu Feng Teng Li Weina Zhang Qing Xia Jianghong Man Tao Zhou Jiayi Chen Ailing Li Xin Pan

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Key Points

MCU is preferentially upregulated in glioblastoma stem cells, indicating its role in cellular metabolism.
The observation is based on Figure S1, which highlights the regulation of MCU in cancer stem cells.
This analysis connects mitochondrial function with acetylation changes in glioblastoma cells.
Investigating MCU may lead to insights into targeting metabolism for glioblastoma treatments.

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Implication

Figure S1 shows MCU is upregulated in glioblastoma stem cells, suggesting a link to acetylation processes.

Abstract

Figure S1 shows that MCU is preferentially upregulated in GSCs.

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Cite This Study

Liu et al. (2025) studied this question.

synapsesocial.com/papers/68d4508931b076d99fa588ab https://doi.org/10.1158/0008-5472.30125839

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Figure S3 from Mitochondrial Calcium Uniporter Links Acetyl-CoA Metabolism and H3K27 Acetylation to Maintain Glioblastoma Stem Cells2025
2Figure S4 from Mitochondrial Calcium Uniporter Links Acetyl-CoA Metabolism and H3K27 Acetylation to Maintain Glioblastoma Stem Cells2025
3Figure S2 from Mitochondrial Calcium Uniporter Links Acetyl-CoA Metabolism and H3K27 Acetylation to Maintain Glioblastoma Stem Cells2025
4Figure S5 from Mitochondrial Calcium Uniporter Links Acetyl-CoA Metabolism and H3K27 Acetylation to Maintain Glioblastoma Stem Cells2025
5Data from Mitochondrial Calcium Uniporter Links Acetyl-CoA Metabolism and H3K27 Acetylation to Maintain Glioblastoma Stem Cells2025

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