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March 1, 2004Journal of HypertensionOpen Access

Antifibrotic effect of Ac-SDKP and angiotensin-converting enzyme inhibition in hypertension

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Key result

Exogenous Ac-SDKP mimicked the anti-inflammatory and antifibrotic effects of ACE inhibitors in Ang II-induced hypertension, independent of hemodynamic effects.

Why the study?

Does Ac-SDKP mimic the anti-inflammatory and antifibrotic effects of ACE inhibitors in rats with Ang II-induced hypertension?

Population

Rats with Angiotensin II-induced hypertension

Comparison

Ac-SDKP or captopril vs Control rats and rats receiving Angiotensin II…

Design

Preclinical

Authors

SRSaman RasoulOCOscar A. CarreteroHPHongmei Peng

Discussion

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Overview

Ac-SDKP effects in Ang II rats extend mechanistic insight; human trials needed before considering targeted therapy.

Structured PICO

Does Ac-SDKP mimic the anti-inflammatory and antifibrotic effects of ACE inhibitors in rats with Ang II-induced hypertension?

P
Population
Rats with Angiotensin II-induced hypertension
I
Intervention
Ac-SDKP (400 or 800 microg/kg per day, s.c.) or captopril (100 mg/kg per day in drinking water)
C
Comparator
Control rats and rats receiving Angiotensin II alone
O
Outcome
Left ventricular cell proliferation, inflammatory cell infiltration, cytokine expression, hypertrophy and fibrosissurrogate

Ac-SDKP mimics the anti-inflammatory and antifibrotic effects of ACE inhibitors in a rat model of hypertension, independent of hemodynamic changes.

Cite This Study

Rasoul et al. (2004) studied Hypertension. Ac-SDKP and captopril vs. Controls and Ang II alone was evaluated on LV cell proliferation, inflammatory cell infiltration, cytokine expression, hypertrophy and fibrosis. Exogenous Ac-SDKP mimicked the anti-inflammatory and antifibrotic effects of ACE inhibitors in Ang II-induced hypertension, independent of hemodynamic effects.

synapsesocial.com/papers/6a16637ac1ea77d8cb623700https://doi.org/10.1097/00004872-200403000-00023
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Also Consider

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