April 1, 1989Molecular and Cellular BiologyOpen Access

An internal regulatory element controls troponin I gene expression.

Q: What is the clinical evidence from this study?

Study design: Other. Population: Skeletal myogenesis. Intervention: Internal regulatory element of the quail troponin I (TnI) gene vs. 5'-flanking regions of other contractile protein genes. Primary outcome: Transcriptional activation during myogenesis.

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Key result

The quail troponin I gene utilizes a unique internal regulatory element to elicit correct myofiber-specific transcriptional activation during myogenesis.

Authors

KYKatherine E. YutzeyPediatric Cardiology RKRobyn L. KlinePurdue University West Lafayette SKStephen F. KoniecznyBoston University

Discussion

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Implication

Suggests coordinated transcriptional control during myogenesis; leaves open extension to cardiac muscle.

PICO

Population

Skeletal myogenesis

Exposure / Comparator

Internal regulatory element of the quail troponin I (TnI) gene vs 5'-flanking regions of other contractile protein genes

Primary Outcome

Transcriptional activation during myogenesis

Cite This Study

Yutzey et al. (1989) studied Skeletal myogenesis. Internal regulatory element of the quail troponin I (TnI) gene vs. 5'-flanking regions of other contractile protein genes was evaluated on Transcriptional activation during myogenesis. The quail troponin I gene utilizes a unique internal regulatory element to elicit correct myofiber-specific transcriptional activation during myogenesis.

synapsesocial.com/papers/6a658fd11a73af1ea1b6a2e1 https://doi.org/10.1128/mcb.9.4.1397

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An internal regulatory element controls troponin I gene expression.

Discussion

Member takes

Implication

PICO

Cite This Study

An internal regulatory element controls troponin I gene expression.

Discussion

Member takes

Implication

PICO

Cite This Study

Also Consider

Also Consider