February 14, 2022Journal of Obstetrics and Gynaecology

MHR was significantly different across the four PCOS phenotypes, with the highest value in phenotype A (13.7 ± 4.9) and the lowest in phenotype D (9.0 ± 1.9).

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Why the study?

To evaluate the degree of inflammation across different polycystic ovary syndrome phenotypes by comparing the monocyte-to-high-density lipoprotein ratio.

Does the monocyte-to-HDL ratio (MHR) differ across different phenotypes of polycystic ovary syndrome (PCOS)?

Population

186 women with PCOS and 59 age-matched healthy women

Comparison

Non-PCOS controls vs four PCOS phenotypes

Design

Case-control study

Authors

CSCenk Soysal İBİsmail Bıyık OİOnur İnce

Discussion

Member takes

Overview

MHR may mark phenotype-specific inflammation in PCOS; leaves open its value for CV risk stratification in observational data.

Structured PICO

Does the monocyte-to-HDL ratio (MHR) differ across different phenotypes of polycystic ovary syndrome (PCOS)?

Population

186 women with polycystic ovary syndrome (PCOS) and 59 age-matched healthy women (Turkish women)

Intervention

PCOS phenotypes (A, B, C, D) based on NIH Expert Panel criteria

Comparator

Non-PCOS control group and comparison between different PCOS phenotypes

Outcome

Monocyte-to-high-density lipoprotein ratio (MHR)surrogate

The monocyte-to-HDL ratio is significantly elevated in the androgenic phenotypes of PCOS, suggesting it may serve as a simple marker for detecting at-risk metabolic phenotypes.

Cite This Study

Soysal et al. (2022) studied this question.

synapsesocial.com/papers/6a6f8cd178a11c550e0945ea https://doi.org/10.1080/01443615.2022.2026309

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February 14, 2022Journal of Obstetrics and Gynaecology

MHR was significantly different across the four PCOS phenotypes, with the highest value in phenotype A (13.7 ± 4.9) and the lowest in phenotype D (9.0 ± 1.9).

View Full Paper

Ask AI

Bookmark

Why the study?

To evaluate the degree of inflammation across different polycystic ovary syndrome phenotypes by comparing the monocyte-to-high-density lipoprotein ratio.

Does the monocyte-to-HDL ratio (MHR) differ across different phenotypes of polycystic ovary syndrome (PCOS)?

Population

186 women with PCOS and 59 age-matched healthy women

Comparison

Non-PCOS controls vs four PCOS phenotypes

Design

Case-control study

Authors

CSCenk Soysal İBİsmail Bıyık OİOnur İnce

Discussion

Member takes

Overview

MHR may mark phenotype-specific inflammation in PCOS; leaves open its value for CV risk stratification in observational data.

Structured PICO

Does the monocyte-to-HDL ratio (MHR) differ across different phenotypes of polycystic ovary syndrome (PCOS)?

Population

186 women with polycystic ovary syndrome (PCOS) and 59 age-matched healthy women (Turkish women)

Intervention

PCOS phenotypes (A, B, C, D) based on NIH Expert Panel criteria

Comparator

Non-PCOS control group and comparison between different PCOS phenotypes

Outcome

Monocyte-to-high-density lipoprotein ratio (MHR)surrogate

The monocyte-to-HDL ratio is significantly elevated in the androgenic phenotypes of PCOS, suggesting it may serve as a simple marker for detecting at-risk metabolic phenotypes.

Cite This Study

Soysal et al. (2022) studied this question.

synapsesocial.com/papers/6a6f8cd178a11c550e0945ea https://doi.org/10.1080/01443615.2022.2026309

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

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