Population
47 subjects comprising 17 with heart failure, 14 with left ventricular hypertrophy, and 16 control subjects
Design
Cross-sectional
Authors
Loading...
Impaired LV systolic function may drive QTc prolongation in cardiac disease; leaves open independent catecholamine effects when function is severely reduced.
In subjects with cardiac disease, QTc interval lengthening is primarily determined by left ventricular systolic performance rather than autonomic function, although catecholamines play an independent role in those with reduced systolic function.
Patrick Davey (2000) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: