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June 1, 1993Molecular and Cellular Biology

Developmental Analysis of Tropomyosin Gene Expression in Embryonic Stem Cells and Mouse Embryos

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Authors

MMMariappan MuthuchamyTexas A&M Health Science Center LPLaura PajakBeckman Coulter Foundation PHPhilip N. HowlesUniversity of Cincinnati

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Implication

Defines cardiac TM isoform expression patterns; leaves open functional roles in contractility with no immediate clinical implications.

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Key points are not available for this paper at this time.

Cite This Study

Muthuchamy et al. (1993) studied this question.

synapsesocial.com/papers/6a705c418031ec7bb1dc480d https://doi.org/10.1128/mcb.13.6.3311-3323.1993

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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Splicing of two alternative exon pairs in beta-tropomyosin pre-mRNA is independently controlled during myogenesis.1992 · 21 citations
2Alpha-tropomyosin gene organization. Alternative splicing of duplicated isotype-specific exons accounts for the production of smooth and striated muscle isoforms.1987 · 175 citations
3Nonmuscle and Muscle Tropomyosin Isoforms Are Expressed from a Single Gene by Alternative RNA Splicing and Polyadenylation1986 · 192 citations
4Rat embryonic fibroblast tropomyosin 1. cDNA and complete primary amino acid sequence.1985 · 40 citations
5Studies on cardiac myofibrillogenesis with antibodies to titin, actin, tropomyosin, and myosin.1988 · 155 citations